Properties of vasoactive‐intestinal‐peptide receptors and β‐adrenoceptors in the murine radiation leukemia‐virus‐induced lymphoma cell line BL/VL3

Abstract
1. Based on radioligand binding and adenylate cyclase activation, functional receptors to vasocative intestinal peptide(VIP)/helodermin, were shown to coexist with .beta.2-adrenoceptors and prostaglandin receptors in membranes from a cultured cloned BL/VL3 cell line of murine T-cell lymphoma induced by a radiation leukemia virus. 2. The relative potency of VIP-related peptides to stimulate adenylate cyclase activity was: helodermin > VIP > peptide histidine isoleucinamide. Five VIP analogs inhibited 125I-iodo-VIP binding and stimulated adenylate cyclase activity, their decreasing order of potency being: VIP > [D-Asp3]VIP > [D-Ser2]VIP > [D-Ala4]VIP = [D-His1]VIP = [D-Phe2]VIP. [D-Phe2]VIP acted as a partial agonist (with an intrinsic activity of 0.1 as compared to that of VIP = 1.0) and competitively inhibited helodermin- and VIP-stimulated adenylate cyclase activity with a similar Ki (0.07-0.10 .mu.M). These data suggest the existence, in the murine T-cell lymphoma, of VIP receptors of the ''helodermin-preferring'' subtype that are coupled to adenylate cyclase.

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