Flt3 mutation activates p21WAF1/CIP1 gene expression through the action of STAT5
- 1 March 2004
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 316 (1) , 85-92
- https://doi.org/10.1016/j.bbrc.2004.02.018
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- The roles of FLT3 in hematopoiesis and leukemiaBlood, 2002
- Analysis of FLT3-activating mutations in 979 patients with acute myelogenous leukemia: association with FAB subtypes and identification of subgroups with poor prognosisBlood, 2002
- FLT3 internal tandem duplication mutations associated with human acute myeloid leukemias induce myeloproliferative disease in a murine bone marrow transplant modelBlood, 2002
- Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathwaysBlood, 2000
- Tandem-duplicated Flt3 constitutively activates STAT5 and MAP kinase and introduces autonomous cell growth in IL-3-dependent cell linesOncogene, 2000
- CDK inhibitors: positive and negative regulators of G1-phase progressionGenes & Development, 1999
- Signal Transduction through MAP Kinase CascadesPublished by Elsevier ,1998
- Tandem duplications of the FLT3 receptor gene are associated with leukemic transformation of myelodysplasiaLeukemia, 1997
- Biochemical Characterization of a Novel KRAS Insertion Mutation from a Human LeukemiaJournal of Biological Chemistry, 1996
- The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinasesCell, 1993