Blockade of human IsK channels expressed in Xenopus oocytes by the novel class III antiarrhythmic NE-10064
- 13 October 1994
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 264 (1) , 33-37
- https://doi.org/10.1016/0014-2999(94)90632-7
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- The Novel Class III Antiarrhythmics NE-10064 and NE-10133 Inhibit Isk Channels Expressed in Xenopus Oocytes and Iks in Guinea Pig Cardiac MyocytesBiochemical and Biophysical Research Communications, 1994
- Effects of Calcium Buffers and Temperature on the β-Receptor-Mediated Regulation of the Slowly Activating Potassium Current (lKS) in Guinea Pig Cardiac MyocytesCellular Physiology and Biochemistry, 1994
- Protection Against Programmed Electrical Stimulation-Induced Ventricular Tachycardia and Sudden Cardiac Death by NE-10064, a Class III Antiarrhythmic DrugJournal of Cardiovascular Pharmacology, 1993
- Effects of [Ca2+]i and temperature on minK channels expressed in Xenopus oocytesFEBS Letters, 1993
- Expression of a minimal K+ channel protein in mammalian cells and immunolocalization in guinea pig heart.Circulation Research, 1993
- The protein IsK is a dual activator of K+ and CI− channelsNature, 1993
- Spectroscopic and functional characterization of the putative transmembrane segment of the minK potassium channelBiochemistry, 1993
- MinK Channels: A Minimal Channel Protein with a Maximal ImpactCellular Physiology and Biochemistry, 1993
- An Amino Acid Mutation in a Potassium Channel That Prevents Inhibition by Protein Kinase CScience, 1992
- Channel specificity in antiarrhythmic drug action. Mechanism of potassium channel block and its role in suppressing and aggravating cardiac arrhythmias.Circulation, 1990