Modulation of ligand binding to components of the GABAA receptor complex by ammonia: implications for the pathogenesis of hyperammonemic syndromes
- 13 May 1996
- journal article
- Published by Elsevier in Brain Research
- Vol. 720 (1-2) , 35-44
- https://doi.org/10.1016/0006-8993(96)00104-7
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- The action of ammonia on astrocyte glycogen and glycogenolysisMolecular and Chemical Neuropathology, 1993
- The contribution of endogenous benzodiazepine receptor ligands to the pathogenesis of hepatic encephalopathySynapse, 1991
- Changes in Brain Metabolism During Hyperammonemia and Acute Liver Failure: Results of A Comparative 1H–Nmr Spectroscopy and Biochemical InvestigationHepatology, 1990
- Hyperammonemia in Women with a Mutation at the Ornithine Carbamoyltransferase LocusNew England Journal of Medicine, 1990
- Increased brain uptake of γ-aminobutyric acid in a rabbit model of hepatic encephalopathyGastroenterology, 1990
- GABAA Receptor Complex in an Experimental Model of Hepatic Encephalopathy: Evidence for Elevated Levels of an Endogenous Benzodiazepine Receptor LigandJournal of Neurochemistry, 1989
- Preservation of “peripheral” benzodiazepine receptors: Differential effects of freezing on [3H]Ro 5-4864 and [3H]PK 11195 bindingJournal of Pharmacological Methods, 1987
- Ammonia: Key factor in the pathogenesis of hepatic encephalopathyNeurochemical Pathology, 1987
- Ammonia does not selectively block IPSPs in rat hippocampal pyramidal cellsJournal of Neurophysiology, 1983
- Biochemical Mechanisms of Hepatic EncephalopathyClinical Science, 1983