DNA Repair Systems in the Phototrophic Bacterium Rhodobacter Capsulatus
- 1 April 1987
- journal article
- research article
- Published by Microbiology Society in Microbiology
- Vol. 133 (4) , 961-966
- https://doi.org/10.1099/00221287-133-4-961
Abstract
SUMMARY: UV irradiation and mitomycin C exposure trigger a protease-activity-dependent inhibition of cell division in Rhodobacter capsulatus, which begins about 2 h after the treatment is applied. UV irradiation also induces a dose-dependent mutagenesis with a maximal rate between 5 and 10 J m−2, with increased synthesis of a protein of Mr approximately 30000 between 2 and 3 h after UV irradiation. In addition, R. capsulatus has an efficient photoreactivation system that reverses the lethal effects of UV irradiation in the presence of intense visible light.This publication has 2 references indexed in Scilit:
- Evidence that a system similar to the recA system of Escherichia coli exists in Vibrio choleraeMolecular Genetics and Genomics, 1985
- The uvrB gene of Escherichia coli has both lexA-repressed and lexA-independent promotersCell, 1982