The c-myc oncogene is regulated independently of differentiation in myeloid cell lines
- 1 January 1989
- journal article
- research article
- Published by Elsevier in Leukemia Research
- Vol. 13 (8) , 651-659
- https://doi.org/10.1016/0145-2126(89)90053-2
Abstract
No abstract availableThis publication has 44 references indexed in Scilit:
- Transcription interruption may be a common mechanism of c-myc regulation during HL-60 differentiationBiochemical and Biophysical Research Communications, 1988
- Recombinant interferon gamma augments phagocyte superoxide production and X-chronic granulomatous disease gene expression in X-linked variant chronic granulomatous disease.Journal of Clinical Investigation, 1987
- Rapid transcriptional down-regulation of c-myc expression during cyclic adenosine monophosphate-promoted differentiation of leukemic cells.Journal of Clinical Investigation, 1987
- A block to elongation is largely responsible for decreased transcription of c-myc in differentiated HL60 cellsNature, 1986
- Regulation of C-myc expression during growth and differentiation of normal and leukemic human myeloid progenitor cells.Journal of Clinical Investigation, 1986
- Expression of the c-fms proto-oncogene during human monocytic differentiationNature, 1985
- Human C-myc and N-ras expression during induction of HL-60 cellular differentiationBiochemical and Biophysical Research Communications, 1985
- The role of interferon-γ in induction of differentiation of human myeloid leukemia cell lines, ML-1 and HL-60Biochemical and Biophysical Research Communications, 1984
- Gamma interferon induces monocytoid differentiation in the HL-60 cell line.Journal of Clinical Investigation, 1984
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970