Kidney immune cell infiltration and oxidative stress contribute to prenatally programmed hypertension
- 1 November 2005
- journal article
- Published by Elsevier in Kidney International
- Vol. 68 (5) , 2180-2188
- https://doi.org/10.1111/j.1523-1755.2005.00674.x
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Oxidant stress in kidneys of spontaneously hypertensive rats involves both oxidase overexpression and loss of extracellular superoxide dismutaseAmerican Journal of Physiology-Renal Physiology, 2004
- l -Arginine Reverses p47phox and gp91phox Expression Induced by High Salt in Dahl RatsHypertension, 2003
- In Salt-Sensitive Hypertension, Increased Superoxide Production Is Linked to Functional Upregulation of Angiotensin IIHypertension, 2003
- Oxidative Stress in Dahl Salt-Sensitive HypertensionHypertension, 2003
- Renal function and angiotensin AT1 receptor expression in young rats following intrauterine exposure to a maternal low-protein dietClinical Science, 2003
- Antioxidant Activities and Oxidative Stress Byproducts in Human HypertensionHypertension, 2003
- Enhanced Oxidative Stress As a Potential Mechanism Underlying the Programming of Hypertension In UteroJournal of Cardiovascular Pharmacology, 2002
- Antihypertensive treatment in early postnatal life modulates prenatal dietary influences upon blood pressure in the ratClinical Science, 2000
- NF-κB Inhibition Ameliorates Angiotensin II–Induced Inflammatory Damage in RatsHypertension, 2000
- Two-Week Administration of Tempol Attenuates Both Hypertension and Renal Excretion of 8-Iso Prostaglandin F 2αHypertension, 1999