L-arginine depletion inhibits glomerular nitric oxide synthesis and exacerbates rat nephrotoxic nephritis
- 1 April 1996
- journal article
- Published by Elsevier in Kidney International
- Vol. 49 (4) , 1090-1096
- https://doi.org/10.1038/ki.1996.158
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- l‐Arginine depletion by arginase reduces nitric oxide production in endotoxic shock: an electron paramagnetic resonance studyFEBS Letters, 1995
- Expression of the gene for inducible nitric oxide synthase in experimental glomerulonephritis in the ratClinical and Experimental Immunology, 1994
- Induction of nitric oxide synthase in rat immune complex glomerulonephritisKidney International, 1994
- Arginine supply for nitric oxide synthesis and arginase is mainly exogenous in elicited murine and rat macrophagesLife Sciences, 1994
- Arginase is a major pathway of L-arginine metabolism in nephritic glomeruliKidney International, 1992
- Glomeruli synthesize nitrite in active Heymann nephritis; the source is infiltrating macrophagesKidney International, 1991
- Glomeruli synthesize nitrite in experimental nephrotoxic nephritisKidney International, 1990
- Interleukin 1 and tumor necrosis factor stimulate cGMP formation in rat renal mesangial cellsFEBS Letters, 1990
- Aggravation of experimental glomerulonephritis by superimposed clip hypertensionKidney International, 1982
- A mononuclear cell component in experimental immunological glomerulonephritis.The Journal of Experimental Medicine, 1978