The INT1 oncogene is not rearranged or amplified in lipomas with structural chromosomal abnormalities of 12q13–15
- 1 October 1989
- journal article
- research article
- Published by Elsevier in Cancer Genetics and Cytogenetics
- Vol. 42 (1) , 143-146
- https://doi.org/10.1016/0165-4608(89)90017-4
Abstract
No abstract availableThis publication has 14 references indexed in Scilit:
- Three major cytogenetic subgroups can be identified among chromosomally abnormal solitary lipomasHuman Genetics, 1988
- A specific translocation, t(12;14)(q14–15; q23–24), characterizes a subgroup of uterine leiomyomasCancer Genetics and Cytogenetics, 1988
- Chromosome localization of the human oncogene INTl to 12ql3 by in situ hybridizationCytogenetic and Genome Research, 1988
- Karyotypic rearrangements in 20 uterine leiomyomasCytogenetic and Genome Research, 1988
- Amplification of the human putative oncogene INT1 in primary retinoblastoma tumorsCytogenetic and Genome Research, 1988
- Lipomas have characteristic structural chromosomal rearrangements of 12q13‐q14International Journal of Cancer, 1987
- Cytogenetic studies of adipose tissue tumors. II. Recurrent reciprocal translocation t(12;16)(q13;p11) in myxoid liposarcomasCancer Genetics and Cytogenetics, 1986
- A retrovirus vector expressing the putative mammary oncogene int-1 causes partial transformation of a mammary epithelial cell lineCell, 1986
- Molecular cloning and chromosomal assignment of the human homolog of int-1, a mouse gene implicated in mammary tumorigenesis.Molecular and Cellular Biology, 1984
- Activation of cellular gene by mouse mammary tumour virus may occur early in mammary tumour developmentNature, 1984