Status epilepticus-induced hippocampal damage is modulated by glucose availability
Open Access
- 1 February 1989
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 97 (1-2) , 157-162
- https://doi.org/10.1016/0304-3940(89)90156-0
Abstract
No abstract availableThis publication has 21 references indexed in Scilit:
- Limbic seizure and brain damage produced by kainic acid: Mechanisms and relevance to human temporal lobe epilepsyPublished by Elsevier ,2003
- Receptors, ion channels and synaptic potentials underlying the integrative actions of excitatory amino acidsTrends in Neurosciences, 1987
- Neuronal calcium homeostasisTrends in Neurosciences, 1987
- Influence of the plasma glucose level on brain damage after systemic kainic acid injection in the ratActa Neuropathologica, 1986
- Influence of Reduced Oxygen Availability on Cerebral Metabolic Changes during Bicuculline-Induced Seizures in RatsJournal of Cerebral Blood Flow & Metabolism, 1985
- Cellular Origin of Ischemia‐Induced Glutamate Release from Brain Tissue In Vivo and In VitroJournal of Neurochemistry, 1985
- The role of brain edema in epileptic brain damage induced by systemic kainic acid injectionNeuroscience, 1984
- Kainate, N-methylaspartate and other excitatory amino acids increase calcium influx into rat brain cortex cells in vitroNeuroscience Letters, 1983
- Synaptic localization of kainic acid binding sitesNature, 1980
- Hippocampal seizures and failure of inhibitionCanadian Journal of Physiology and Pharmacology, 1979