Fibrinolytic study in a homozygous protein C deficient patient
- 1 May 1986
- journal article
- research article
- Published by Elsevier in Thrombosis Research
- Vol. 42 (3) , 313-322
- https://doi.org/10.1016/0049-3848(86)90260-4
Abstract
No abstract availableThis publication has 37 references indexed in Scilit:
- Neonatal purpura fulminans: a genetic disorder related to the absence of protein C in bloodBlood, 1985
- Severe homozygous protein C deficiencyThe Journal of Pediatrics, 1984
- Homozygous Protein C Deficiency Manifested by Massive Venous Thrombosis in the NewbornNew England Journal of Medicine, 1984
- Severe Inherited “Homozygous” Protein C Deficiency in a Newborn InfantThrombosis and Haemostasis, 1984
- INHERITED PROTEIN C DEFICIENCY AND COUMARIN-RESPONSIVE CHRONIC RELAPSING PURPURA FULMINANS IN A NEWBORN INFANTThe Lancet, 1983
- Activation of Protein C In VivoJournal of Clinical Investigation, 1982
- Mechanism of action of human activated protein C, a thrombin-dependent anticoagulant enzymeBlood, 1982
- Generation of fibrinolytic activity by infusion of activated protein C into dogs.Journal of Clinical Investigation, 1981
- Deficiency of protein C inhibitor in combined factor V/VIII deficiency disease.Journal of Clinical Investigation, 1980
- Anticoagulant properties of bovine plasma protein C following activation by thrombinBiochemistry, 1977