Alteration of neuronal calcium homeostasis and excitotoxic vulnerability by chronic depolarization
- 20 June 1994
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 648 (2) , 291-295
- https://doi.org/10.1016/0006-8993(94)91129-0
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Cell-permeant Ca2+ chelators reduce early excitotoxic and ischemic neuronal injury in vitro and in vivoNeuron, 1993
- Secondary Ca2+ overload indicates early neuronal injury which precedes staining with viability indicatorsBrain Research, 1993
- Stable transfection of calbindin-D28k into the GH3 cell line alters calcium currents and intracellular calcium homeostasisNeuron, 1992
- Membrane depolarization and the expression of glutamate receptors in cerebellar granule cellsEuropean Journal of Pharmacology, 1991
- Dantrolene Prevents Glutamate Cytotoxicity and Ca2+ Release from Intracellular Stores in Cultured Cerebral Cortical NeuronsJournal of Neurochemistry, 1991
- Quin2 protects against neuronal cell death due to Ca2+ overloadBrain Research, 1990
- The effects of potassium‐induced depolarization, glutamate receptor antagonists and N‐methyl‐d‐aspartate on neuronal survival in cultured neocortex explantsInternational Journal of Developmental Neuroscience, 1990
- Dantrolene suppresses the hyperpolarization or outward current observed during anoxia in hippocampal neuronsCanadian Journal of Physiology and Pharmacology, 1989
- Protection of Dentate Hilar Cells from Prolonged Stimulation by Intracellular Calcium ChelationScience, 1989
- Morphological and biochemical differences expressed in separate dissociated cell cultures of dorsal and ventral halves of the mouse spinal cordBrain Research, 1987