Abstract
Several factors including genetic and environmental play a role in the development of obesity and the metabolic syndrome. The transgenic mouse overexpressing 11β‐hydroxysteroid dehydrogenase (11β‐HSD) develops visceral obesity. However, it remains unclear how a ubiquitously expressed 11β‐HSD1 enzyme affects adipose tissue so much that it would lead to obesity. In this commentary we explore the possibility that increased intracellular availability of reduced co‐factor, NADPH, could exacerbate the enzymatic activity.

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