An AP-1 Binding Sequence Is Essential for Regulation of the Human α2(I) Collagen (COL1A2) Promoter Activity by Transforming Growth Factor-β
Open Access
- 1 February 1996
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 271 (6) , 3272-3278
- https://doi.org/10.1074/jbc.271.6.3272
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Characterization of a GC-rich Region Containing Sp1 Binding Site(s) as a Constitutive Responsive Element of the α2(I) Collagen Gene in Human FibroblastsJournal of Biological Chemistry, 1995
- Overlapping Pathways Mediate the Opposing Actions of Tumor Necrosis Factor-α and Transforming Growth Factor-β on α2(I) Collagen Gene TranscriptionJournal of Biological Chemistry, 1995
- Transcriptional interactions of transforming growth-factor-β with pro-inflammatory cytokinesCurrent Biology, 1993
- Cytokine regulation of metalloproteinase gene expressionJournal of Cellular Biochemistry, 1993
- Minimal DNA sequences that control the cell lineage-specific expression of the pro alpha 2(I) collagen promoter in transgenic mice.The Journal of cell biology, 1992
- jun-B inhibits and c-fos stimulates the transforming and trans-activating activities of c-junCell, 1989
- Jun-B differs in its biological properties from, and is a negative regulator of, c-JunCell, 1989
- NF-κB: A pleiotropic mediator of inducible and tissue-specific gene controlCell, 1989
- Prolonged activation of jun and collagenase genes by tumour necrosis factor-αNature, 1989
- A nuclear factor 1 binding site mediates the transcriptional activation of a type I collagen promoter by transforming growth factor-βCell, 1988