CaM Kinase II-dependent Suppression of Nicotinic Acetylcholine Receptor δ-Subunit Promoter Activity
Open Access
- 1 July 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (28) , 26057-26065
- https://doi.org/10.1074/jbc.m101670200
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- DEVELOPMENT OF THE VERTEBRATE NEUROMUSCULAR JUNCTIONAnnual Review of Neuroscience, 1999
- Synapse-specific and neuregulin-induced transcription require an Ets site that binds GABPα/GABPβGenes & Development, 1998
- Molecular Mechanisms Mediating Synapse-Specific Gene Expression During Development of the Neuromuscular JunctionCanadian Journal of Applied Physiology, 1998
- Role for calcium from the sarcoplasmic reticulum in coupling muscle activity to nicotinic acetylcholine receptor gene expression in ratJournal of Neurobiology, 1998
- Implication of a multisubunit Ets-related transcription factor in synaptic expression of the nicotinic acetylcholine receptorThe EMBO Journal, 1998
- In vivo and in vitro analysis of electricalactivity-dependent expression of muscle acetylcholine receptor genes usingadenovirus.Proceedings of the National Academy of Sciences, 1994
- Protein kinase C couples membrane excitation to acetylcholine receptor gene inactivation in chick skeletal muscleNeuron, 1992
- Regulation of muscle AChR α subunit gene expression by electrical activity: Involvement of protein kinase C and Ca 2+Neuron, 1989
- Calcitonin gene-related peptide and muscle activity regulate acetylcholine receptor alpha-subunit mRNA levels by distinct intracellular pathways.The Journal of cell biology, 1987
- Molecular distinction between fetal and adult forms of muscle acetylcholine receptorNature, 1986