Tumor Necrosis Factor-α Increases Airway Smooth Muscle Oxidants Production through a NADPH Oxidase-like System to Enhance Myosin Light Chain Phosphorylation and Contractility

This publication has 45 references indexed in Scilit:

• Rac1 inhibits TNF‐α‐induced endothelial cell apoptosis: dual regulation by reactive oxygen species
  The FASEB Journal, 2000
• Agonists Trigger G Protein-mediated Activation of the CPI-17 Inhibitor Phosphoprotein of Myosin Light Chain Phosphatase to Enhance Vascular Smooth Muscle Contractility
  Journal of Biological Chemistry, 2000
• Signal transduction by G‐proteins, Rho‐kinase and protein phosphatase to smooth muscle and non‐muscle myosin II
  The Journal of Physiology, 2000
• Requirement for Reactive Oxygen Species in Serum-induced and Platelet-derived Growth Factor-induced Growth of Airway Smooth Muscle
  Journal of Biological Chemistry, 1999
• Tumour necrosis factor‐α activates a calcium sensitization pathway in guinea‐pig bronchial smooth muscle
  The Journal of Physiology, 1999
• Mechanisms involved in the contraction of endothelial cells by hydrogen peroxide
  Free Radical Biology & Medicine, 1999
• Tumour necrosis factor-alpha induces hyperreactivity in tracheal smooth muscle of the guinea-pig in vitro
  European Respiratory Journal, 1998
• Tumour necrosis factor alpha: a polcnlial mediator of asthma
  Clinical and Experimental Allergy, 1995
• Requirement for Generation of H ₂ O ₂ for Platelet-Derived Growth Factor Signal Transduction
  Science, 1995
• The use of diphenylene iodonium and its analogues to investigate the role of the nadph oxidase in the tumoricidal activity of macrophages in vitro
  Free Radical Biology & Medicine, 1991