Effects of opioid compounds on desensitization of the nicotinic response of isolated bovine adrenal chromaffin cells

This publication has 21 references indexed in Scilit:

• Effects of Opioid Peptides Containing the Sequence of Met⁵‐Enkephalin or Leu⁵‐Enkephalin on Nicotine‐Induced Secretion from Bovine Adrenal Chromaffin Cells
  Journal of Neurochemistry, 1986
• Interaction of Opiates with Opioid Binding Sites in the Bovine Adrenal Medulla: I. Interaction with δ and μ Sites
  Journal of Neurochemistry, 1985
• Neonatal capsaicin treatment prevents insulin-stress-induced adrenal catecholamine secretion in vivo: Possible involvement of sensory nerves containing substance P
  Neuroscience Letters, 1984
• Desensitization to Nicotinic Cholinergic Agonists and K⁺, Agents That Stimulate Catecholamine Secretion, in Isolated Adrenal Chromaffin Cells
  Journal of Neurochemistry, 1984
• Opiate Receptor‐Mediated Inhibition of Catecholamine Release in Primary Cultures of Bovine Adrenal Chromaffin Cells
  Journal of Neurochemistry, 1982
• Evidence that Inhibition of Nicotine‐Mediated Catecholamine Secretion from Adrenal Chromaffin Cells by Enkephalin, β‐Endorphin, Dynorphin (1‐13), and Opiates is not Mediated via Specific Opiate Receptors
  Journal of Neurochemistry, 1982
• Studies on the Inhibitory Action of Opiate Compounds in Isolated Bovine Adrenal Chromaffin Cells: Noninvolvement of Stereospecific Opiate Binding Sites
  Journal of Neurochemistry, 1981
• Pharmacological characterization of adrenal paraneurons: substance P and somatostatin as inhibitory modulators of the nicotinic response
  Brain Research, 1979
• Substance P inhibits nicotinic activation of chromaffin cells
  Nature, 1979
• A QUANTITATIVE ANALYSIS OF THE INFLUENCE OF EXTERNAL CALCIUM AND MAGNESIUM CONCENTRATIONS ON ACETYLCHOLINEINDUCED ADRENALINE RELEASE IN RAT ADRENAL GLAND PERFUSED IN AN ANTIDROMIC OR ORTHODROMIC DIRECTION
  The Japanese Journal of Physiology, 1977