Nitric Oxide-induced Transcriptional Up-regulation of Protective Genes by Nrf2 via the Antioxidant Response Element Counteracts Apoptosis of Neuroblastoma Cells
Open Access
- 1 May 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (19) , 20096-20107
- https://doi.org/10.1074/jbc.m312492200
Abstract
No abstract availableKeywords
This publication has 68 references indexed in Scilit:
- JNK-dependent Phosphorylation of c-Jun on Serine 63 Mediates Nitric Oxide-induced Apoptosis of Neuroblastoma CellsJournal of Biological Chemistry, 2004
- Nitric Oxide Protects Neuroblastoma Cells from Apoptosis Induced by Serum Deprivation through cAMP-response Element-binding Protein (CREB) ActivationJournal of Biological Chemistry, 2002
- Does nitric oxide modulate mitochondrial energy generation and apoptosis?Nature Reviews Molecular Cell Biology, 2002
- Cobalt Induces Heme Oxygenase-1 Expression by a Hypoxia-inducible Factor-independent Mechanism in Chinese Hamster Ovary CellsJournal of Biological Chemistry, 2001
- Nrf2-Dependent Activation of the Antioxidant Responsive Element by tert-Butylhydroquinone Is Independent of Oxidative Stress in IMR-32 Human Neuroblastoma CellsBiochemical and Biophysical Research Communications, 2001
- Superoxide enhances interleukin 1β–mediated transcription of the hepatocyte-inducible nitric oxide synthase geneGastroenterology, 2000
- Nitric oxide (NO): an effector of apoptosisCell Death & Differentiation, 1999
- Mechanisms for Redox Control of Gene ExpressionAnnual Review of Microbiology, 1999
- Transcriptional control by protein phosphorylation: signal transmission from the cell surface to the nucleusCurrent Biology, 1995
- Enhanced NAD(P)H:Quinone Reductase Activity Prevents Glutamate Toxicity Produced by Oxidative StressJournal of Neurochemistry, 1991