NMDA antagonists: Lack of protective effect against hypoxic damage in CA1 region of hippocampal slices
- 1 June 1988
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 89 (2) , 187-192
- https://doi.org/10.1016/0304-3940(88)90379-5
Abstract
No abstract availableThis publication has 15 references indexed in Scilit:
- Extracellular overflow of glutamate, aspartate, GABA and taurine in the cortex and basal ganglia of fetal lambs during hypoxia-ischemiaNeuroscience Letters, 1987
- Blockade of excitatory amino acid receptors protects anoxic hippocampal slicesNeuroscience, 1987
- Chlorpromazine protects brain tissue in hypoxia by delaying spreading depression-mediated calcium influxBrain Research, 1986
- Barbiturate protection against hypoxic neuronal damage in vitroJournal of Neurosurgery, 1986
- Glutamate and the pathophysiology of hypoxic–ischemic brain damageAnnals of Neurology, 1986
- N-methyl-D-aspartate receptor antagonists reduce synaptic excitation in the hippocampusJournal of Neuroscience, 1986
- Ischemia-Induced Shift of Inhibitory and Excitatory Amino Acids from Intra- to Extracellular CompartmentsJournal of Cerebral Blood Flow & Metabolism, 1985
- Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral MicrodialysisJournal of Neurochemistry, 1984
- Synaptic release of excitatory amino acid neurotransmitter mediates anoxic neuronal deathJournal of Neuroscience, 1984
- Mechanisms involved in irreversible anoxic damage to the in vitro rat hippocampal sliceThe Journal of Physiology, 1982