Suppression of Ras-mediated NIH3T3 transformation by p19ARF does not involve alterations of cell growth properties
- 25 March 1999
- journal article
- Published by Springer Nature in Oncogene
- Vol. 18 (12) , 2157-2162
- https://doi.org/10.1038/sj.onc.1202532
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 16 references indexed in Scilit:
- Functional and physical interactions of the ARF tumor suppressor with p53 and Mdm2Proceedings of the National Academy of Sciences, 1998
- Functional analysis of wild-type and malignant glioma derived CDKN2Aβ alleles: Evidence for an RB-independent growth suppressive pathwayOncogene, 1997
- Regulation of p53 stability by Mdm2Nature, 1997
- Mdm2 promotes the rapid degradation of p53Nature, 1997
- Cancer-associated mutations at the INK4a locus cancel cell cycle arrest by p16 INK4a but not by the alternative reading frame protein p19 ARFProceedings of the National Academy of Sciences, 1997
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- Inhibition of Ras-Induced Proliferation and Cellular Transformation by p16 INK4Science, 1995
- Mutations and altered expression of p16INK4 in human cancer.Proceedings of the National Academy of Sciences, 1994
- Analysis of the p16 gene (CDKN2) as a candidate for the chromosome 9p melanoma susceptibility locusNature Genetics, 1994
- A Cell Cycle Regulator Potentially Involved in Genesis of Many Tumor TypesScience, 1994