Toward Understanding the Neuronal Pathogenesis of Aspartylglucosaminuria: Expression of Aspartylglucosaminidase in Brain during Development
- 31 August 1999
- journal article
- Published by Elsevier in Molecular Genetics and Metabolism
- Vol. 67 (4) , 294-307
- https://doi.org/10.1006/mgme.1999.2872
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Primary Folding of AspartylglucosaminidaseJournal of Biological Chemistry, 1996
- Three-dimensional structure of human lysosomal aspartylglucosaminidaseNature Structural & Molecular Biology, 1995
- Molecular Cloning, Chromosomal Assignment, and Expression of the Mouse Aspartylglucosaminidase GeneGenomics, 1995
- Deletion of the C-terminal end of aspartylgiucosaminidase resulting in a lysosomal accumulation disease: evidence for a unique genomic rearrangementHuman Molecular Genetics, 1995
- Myelin basic protein mRNA translocation in oligodendrocytes is inhibited by astrocytes in vitroJournal of Neuroscience Research, 1993
- Expression of aspartylglucosaminidase in human tissues from normal individuals and aspartylglucosaminuria patients.Journal of Histochemistry & Cytochemistry, 1993
- Regional expression of myelin protein genes in the developing mouse brain: In situ hybridization studiesJournal of Neuroscience Research, 1988
- Differentiation of Purkinje cells and their relationship to other components of developing cerebellar cortex in manJournal of Comparative Neurology, 1976
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976