Increased severity of glomerulonephritis in C-C chemokine receptor 2 knockout mice
- 1 January 2000
- journal article
- Published by Elsevier in Kidney International
- Vol. 57 (1) , 129-136
- https://doi.org/10.1046/j.1523-1755.2000.00848.x
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Monocyte chemoattractant protein-1 promotes macrophage-mediated tubular injury, but not glomerular injury, in nephrotoxic serum nephritisJournal of Clinical Investigation, 1999
- Impaired monocyte migration and reduced type 1 (Th1) cytokine responses in C-C chemokine receptor 2 knockout mice.Journal of Clinical Investigation, 1997
- Monocyte chemoattractant protein-1 mediates monocyte/macrophage influx in anti-thymocyte antibody-induced glomerulonephritisKidney International, 1997
- Production of the novel C-C chemokine MCP-4 by airway cells and comparison of its biological activity to other C-C chemokines.Journal of Clinical Investigation, 1997
- Chemokines and renal diseaseKidney International, 1997
- Murine Monocyte Chemoattractant Protein (MCP)-5: A Novel CC Chemokine That Is a Structural and Functional Homologue of Human MCP-1The Journal of Experimental Medicine, 1997
- Differential expression of macrophage inflammatory protein-2 and monocyte chemoattractant protein-1 in experimental glomerulonephritisKidney International, 1996
- Monocyte chemotactic protein-1 (MCP-1), -2, and -3 are chemotactic for human T lymphocytes.Journal of Clinical Investigation, 1995
- Monocyte chemotactic protein 3 is a most effective basophil- and eosinophil-activating chemokine.The Journal of Experimental Medicine, 1994
- The susceptibility of experimental glomerulonephritis in six different strains of mice.Journal of Pharmacobio-Dynamics, 1985