Abstract
Asterixis is usually bilateral. Occasionally, it may appear unilaterally. Hemiasterixis following infarction of the contralateral midbrain and thalamus is described. Mechanisms of asterixis and brain lesions associated with its unilateral appearance are reviewed. The mesodiencephalon is frequently implicated by the lesion either directly or indirectly, suggesting the importance of this structure in posture regulation.

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