Large-cell anaplastic lymphoma-specific translocation (t[2;5] [p23;q35]) in Hodgkin's disease: indication of a common pathogenesis?
- 1 January 1995
- journal article
- Published by Elsevier in The Lancet
- Vol. 345 (8942) , 87-90
- https://doi.org/10.1016/s0140-6736(95)90061-6
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Fusion of a Kinase Gene, ALK , to a Nucleolar Protein Gene, NPM , in Non-Hodgkin's LymphomaScience, 1994
- Localization of two tyrosine kinase receptor genes with respect to the 5q35 chromosomal breakpoint of Ki‐1 lymphoma cell linesGenes, Chromosomes and Cancer, 1993
- Diagnosis and Grading of Nodular Sclerosing Hodgkin's Disease: A Study of 2190 PatientsPublished by Elsevier ,1992
- Signal transduction by receptors with tyrosine kinase activityPublished by Elsevier ,1990
- CD30‐positive large cell lymphomas (‘Ki‐1 lymphoma’) are associated with a chromosomal translocation involving 5q35British Journal of Haematology, 1990
- A chromosomal basis of lymphoid malignancy in manEuropean Journal of Biochemistry, 1989
- Demonstration of monoclonal EBV genomes in Hodgkin's disease and Ki-1- positive anaplastic large cell lymphoma by combined Southern blot and in situ hybridization [see comments]Blood, 1989
- Alternative forms of the BCR-ABL oncogene have quantitatively different potencies for stimulation of immature lymphoid cells.Molecular and Cellular Biology, 1989
- Ki-1 Positive Large Cell LymphomaThe American Journal of Surgical Pathology, 1988
- COMMON ACTIVATED HELPER-T-CELL ORIGIN FOR LYMPHOMATOID PAPULOSIS, MYCOSIS FUNGOIDES, AND SOME TYPES OF HODGKIN'S DISEASEThe Lancet, 1985