Transcription activities at 8-oxoG lesions in DNA
- 6 July 2004
- journal article
- Published by Elsevier in DNA Repair
- Vol. 3 (11) , 1457-1468
- https://doi.org/10.1016/j.dnarep.2004.06.008
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous oxidative DNA base damageOncogene, 2002
- The Cockayne Syndrome Group B Gene Product Is Involved in General Genome Base Excision Repair of 8-Hydroxyguanine in DNAJournal of Biological Chemistry, 2001
- Effect of Thymine Glycol on Transcription Elongation by T7 RNA Polymerase and Mammalian RNA Polymerase IIJournal of Biological Chemistry, 2001
- RETRACTED: Transcription-Coupled Repair of 8-oxoGuanineCell, 2000
- Purification of the Transcription/Repair Factor TFIIH and Evaluation of Its Associated Activitiesin VitroMethods, 1997
- Cloning of a human homolog of the yeast OGG1 gene that is involved in the repair of oxidative DNA damageOncogene, 1997
- Inactivation of OGG1 increases the incidence of G · C→T · A transversions in Saccharomyces cerevisiae : evidence for endogenous oxidative damage to DNA in eukaryotic cellsMolecular Genetics and Genomics, 1997
- Oxidative DNA Lesions as Blocks to in Vitro Transcription by Phage T7 RNA PolymeraseAnnals of the New York Academy of Sciences, 1994
- Insertion of specific bases during DNA synthesis past the oxidation-damaged base 8-oxodGNature, 1991
- Cyclobutane pyrimidine dimers and (6-4) photoproducts block polymerization by DNA polymerase IBiochemistry, 1985