NAAG fails to antagonize synaptic and extrasynaptic NMDA receptors in cerebellar granule neurons
- 31 March 2004
- journal article
- Published by Elsevier in Neuropharmacology
- Vol. 46 (4) , 490-496
- https://doi.org/10.1016/j.neuropharm.2003.10.007
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Glutamate carboxypeptidase II inhibition protects motor neurons from death in familial amyotrophic lateral sclerosis modelsProceedings of the National Academy of Sciences, 2003
- Circuit analysis of NMDAR hypofunction in the hippocampus, in vitro, and psychosis of schizophrenia†Hippocampus, 2001
- Unusual spectral energy distribution of a galaxy previously reported to be at redshift 6.68Nature, 2000
- Activation of mGluRII induces LTD via activation of protein kinase A and protein kinase C in the dentate gyrus of the hippocampus in vitroNeuropharmacology, 1999
- Neuroprotective activity of N-acetylaspartylglutamate in cultured cortical cellsNeuroscience, 1998
- A Direct Comparison of the Single-Channel Properties of Synaptic and Extrasynaptic NMDA ReceptorsJournal of Neuroscience, 1997
- Glutamate and N-acetylaspartylglutamate block HVA calcium currents in frog olfactory bulb interneurons via an mGluR2/3-like receptorJournal of Neurophysiology, 1996
- NMDA-dependent modulation of CA1 local circuit inhibitionJournal of Neuroscience, 1996
- N‐Acetylaspartylglutamate Selectively Inhibits Neuronal Responses to N‐Methyl‐d‐Aspartic Acid In VitroJournal of Neurochemistry, 1994
- The role of depolarization in the survival and differentiation of cerebellar granule cells in cultureJournal of Neuroscience, 1987