The mechanism of actions of 3-(5′-(hydroxymethyl-2′-furyl)-1-benzyl indazole (YC-1) on Ca2+-activated K+ currents in GH3 lactotrophs
- 6 July 2000
- journal article
- Published by Elsevier in Neuropharmacology
- Vol. 39 (10) , 1788-1799
- https://doi.org/10.1016/s0028-3908(00)00025-3
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Mechanism of YC-1-Induced Activation of Soluble Guanylyl CyclaseMolecular Pharmacology, 1998
- Calcium Modulation of Ligand Affinity in the Cyclic GMP–gated Ion Channels of Cone PhotoreceptorsThe Journal of general physiology, 1997
- The cross-talk between nitric oxide and Ca2+: a story with a complex past and a promising futureTrends in Pharmacological Sciences, 1997
- Endothelin Activates Large-Conductance K+ Channels in Rat Lactotrophs: Reversal by Long-Term Exposure to Dopamine AgonistEndocrinology, 1997
- Somatostatin Stimulates BKCa Channels in Rat Pituitary Tumor Cells through Lipoxygenase Metabolites of Arachidonic AcidNeuropharmacology, 1996
- cGMP-mediated effects on the physiology of bovine and human retinal Müller (glial) cells.The Journal of Physiology, 1996
- Nitric Oxide Inhibits the Positive Inotropic Response to β-Adrenergic Stimulation in Humans With Left Ventricular DysfunctionCirculation, 1995
- Role of nitric oxide in the regulation of myocardial functionProgress in Cardiovascular Diseases, 1995
- Biosynthesis and Metabolism of Endothelium-Derived Nitric OxideAnnual Review of Pharmacology and Toxicology, 1990
- Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patchesPflügers Archiv - European Journal of Physiology, 1981