Effects of the carboxyl‐terminal fragment of Alzheimer's amyloid precursor protein and amyloid β ‐peptide on the production of cytokines and nitric oxide in glial cells

This publication has 43 references indexed in Scilit:

• Increased plasma levels of interleukin-1, interleukin-6 and α-1-antichymotrypsin in patients with Alzheimer's disease: peripheral inflammation or signals from the brain?
  Journal of Neuroimmunology, 2000
• Nitric Oxide Synthase Inhibition Delays Axonal Degeneration and Promotes the Survival of Axotomized Retinal Ganglion Cells
  Experimental Neurology, 1999
• Nitric oxide and cell death
  Biochimica et Biophysica Acta (BBA) - Bioenergetics, 1999
• Potential Role of the JNK/SAPK Signal Transduction Pathway in the Induction of iNOS by TNF-α
  Biochemical and Biophysical Research Communications, 1998
• The Intracellular Component of Cellular 3‐(4,5‐Dimethylthiazol‐2‐yl)‐2,5‐Diphenyltetrazolium Bromide (MTT) Reduction Is Specifically Inhibited by β‐Amyloid Peptides
  Journal of Neurochemistry, 1995
• Bacterial expression, purification of full length and carboxyl terminal fragment of alzheimer amyloid precursor protein and their proteolytic processing by thrombin
  Life Sciences, 1994
• Secretory cleavage site of Alzheimer amyloid precursor protein is heterogeneous in Down's syndrome brain
  FEBS Letters, 1994
• Altered processing characteristics of β-amyloid-containing peptides in cytosol and in media of familial Alzheimer's disease cells
  Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1994
• Formation of amyloid-like fibrils in COS cells overexpressing part of the Alzheimer amyloid protein precursor
  Nature, 1990
• Preparation of separate astroglial and oligodendroglial cell cultures from rat cerebral tissue.
  The Journal of cell biology, 1980