Modulation of Nitric Oxide-Induced Apoptotic Death of HL-60 Cells by Protein Kinase C and Protein Kinase A through Mitogen-Activated Protein Kinases and CPP32-like Protease Pathways
- 1 May 1999
- journal article
- Published by Elsevier in Cellular Immunology
- Vol. 194 (1) , 36-46
- https://doi.org/10.1006/cimm.1999.1480
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Role of nitric oxide synthesis in macrophage antimicrobial activityPublished by Elsevier ,2003
- Nitric Oxide Modulates the c-Jun N-terminal Kinase/Stress-Activated Protein Kinase Activity through Activating c-Jun N-Terminal Kinase KinaseBiochemistry, 1997
- Molecular Mechanisms of Apoptosis in HL-60 Cells Induced by a Nitric Oxide-Releasing CompoundFree Radical Research, 1997
- Differential Activation of Mitogen-activated Protein Kinases by Nitric Oxide-related SpeciesJournal of Biological Chemistry, 1996
- Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death.The Journal of cell biology, 1996
- Posttranslational Modification of Glyceraldehyde-3-phosphate Dehydrogenase by S-Nitrosylation and Subsequent NADH AttachmentJournal of Biological Chemistry, 1996
- Parallel signal processing among mammalian MAPKsTrends in Biochemical Sciences, 1995
- A MAP Kinase Targeted by Endotoxin and Hyperosmolarity in Mammalian CellsScience, 1994
- The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzymeCell, 1993
- DNA Deaminating Ability and Genotoxicity of Nitric Oxide and its ProgenitorsScience, 1991