Determinants of Inhibition of Transiently Expressed Voltage-gated Calcium Channels by ω-Conotoxins GVIA and MVIIA
Open Access
- 1 May 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (22) , 20171-20178
- https://doi.org/10.1074/jbc.m300581200
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Actions of intrathecal ω-conotoxins CVID, GVIA, MVIIA, and morphine in acute and neuropathic pain in the ratEuropean Journal of Pharmacology, 2002
- Inhibition of transiently expressed low- and high-voltage-activated calcium channels by trivalent metal cations.The Journal of Membrane Biology, 2002
- Intractable Delirium Associated With Ziconotide Successfully Treated With Electroconvulsive TherapyPsychosomatics, 2002
- Suppression of inflammatory and neuropathic pain symptoms in mice lacking the N-type Ca2+ channelThe EMBO Journal, 2001
- Residue Gly1326 of the N-type Calcium Channel α1B Subunit Controls Reversibility of ω-Conotoxin GVIA and MVIIA BlockJournal of Biological Chemistry, 2001
- An evaluation of intrathecal ziconotide for the treatment of chronic painExpert Opinion on Investigational Drugs, 2000
- Irreversible and reversible blockade of IMR32 calcium channel currents by synthetic MVIIA and iodinated MVIIC ?-conopeptidesPflügers Archiv - European Journal of Physiology, 1995
- Structural determinants of the blockade of N-type calcium channels by a peptide neurotoxinNature, 1994
- Calcium Channel Subtypes in Rat Brain: Biochemical Characterization of the High-Affinity Receptors for ω-Conopeptides SNX-230 (Synthetic MVIIC), SNX-183 (SVIB), and SNX-111 (MVIIA)Molecular and Cellular Neuroscience, 1994
- CALCIUM CHANNEL DIVERSITY AND NEUROTRANSMITTER RELEASE: The ω-Conotoxins and ω-AgatoxinsAnnual Review of Biochemistry, 1994