Prostaglandin E1-mediated, cyclic AMP-independent, down-regulation of Gs alpha in neuroblastoma x glioma hybrid cells.

This publication has 41 references indexed in Scilit:

• Biphasic regulation of adenylate cyclase by cholera toxin in neuroblastoma × glioma hybrid cells is due to the activation and subsequent loss of the α subunit of the stimulatory GTP binding proteins (Gs)
  Cellular Signalling, 1990
• Gi2 mediates alpha 2-adrenergic inhibition of adenylyl cyclase in platelet membranes: in situ identification with G alpha C-terminal antibodies.
  Proceedings of the National Academy of Sciences, 1989
• Guanine nucleotide binding proteins and cellular control
  Current Opinion in Cell Biology, 1989
• A mutation that prevents GTP-dependent activation of the α chain of Gs
  Nature, 1988
• Chronic ethanol causes heterologous desensitization of receptors by reducing αsmessenger RNA
  Nature, 1988
• G PROTEINS: TRANSDUCERS OF RECEPTOR-GENERATED SIGNALS
  Annual Review of Biochemistry, 1987
• Prostacyclin analogues reduce ADP‐ribosylation of the α‐subunit of the regulatory G_s‐protein and diminish adenosine (A₂) responsiveness of platelets
  British Journal of Pharmacology, 1987
• Molecular mechanisms of receptor desensitization using the β-adrenergic receptor-coupled adenylate cyclase system as a model
  Nature, 1985
• Structural mutations in a mouse immunoglobulin light chain resulting in failure to be secreted
  Cell, 1980
• A highly sensitive adenylate cyclase assay
  Analytical Biochemistry, 1974