Case report of pallido‐pyramidal disease with supplementary motor area involvement
- 26 July 2001
- journal article
- research article
- Published by Wiley in Movement Disorders
- Vol. 16 (4) , 762-764
- https://doi.org/10.1002/mds.1126
Abstract
An C-flumazenil positron emission tomography (PET) study in a patient with pallido-pyramidal disease revealed a marked decrease in benzodiazepine-receptor density in the precentral gyrus cortex and the mesial frontal cortex. We suggest that, in addition to dysfunction of basal ganglia-dependent systems, degeneration of the supplementary motor area could also be involved in the patient's bradykinesia. © 2001 Movement Disorder Society.Keywords
This publication has 8 references indexed in Scilit:
- Striatal dopaminergic denervation in Pallidopyramidal disease demonstrated by positron emission tomographyAnnals of Neurology, 1995
- Positron Emission Tomography Measures of Benzodiazepine Binding in Alzheimer's DiseaseArchives of Neurology, 1995
- Familial levodopa‐responsive parkinsonian‐pyramidal syndromeMovement Disorders, 1994
- Axial motor disturbances after hypoxic lesions of the globus pallidusMovement Disorders, 1993
- Impaired activation of the supplementary motor area in Parkinson's disease is reversed when akinesia is treated with apomorphineAnnals of Neurology, 1992
- Simple and complex movements in a patient with infarction of the right supplementary motor areaMovement Disorders, 1986
- Pallido-pyramidal syndrome treated with levodopa.Journal of Neurology, Neurosurgery & Psychiatry, 1975
- PALLIDO-PYRAMIDAL DISEASEJournal of Neuropathology and Experimental Neurology, 1954