Noradrenergic mechanisms appear not to be involved in cocaine-induced seizures and lethality
- 1 January 1990
- journal article
- research article
- Published by Elsevier in Life Sciences
- Vol. 47 (4) , 353-359
- https://doi.org/10.1016/0024-3205(90)90594-h
Abstract
No abstract availableThis publication has 16 references indexed in Scilit:
- Are there multiple imidazoline binding sites?Trends in Pharmacological Sciences, 1989
- POTENTIAL TOXIC INTERACTIONS OF COCAINE AND MAZINDOLThe Lancet, 1989
- Interactions of alpha2-adrenoceptor antagonists with medetomidine and with ethanol in a holeboard testNeuropharmacology, 1989
- Desipramine Facilitation of Initial Cocaine AbstinenceArchives of General Psychiatry, 1989
- Lethal effects of cocaine are reduced by the dopamine-1 receptor antagonist SCH 23390 but not by haloperidolLife Sciences, 1989
- Studies on RX 781094: a selective, potent and specific antagonist of α2‐adrenoceptorsBritish Journal of Pharmacology, 1983
- Antagonism of Intravenous Cocaine Lethality in Nonhuman PrimatesClinical Toxicology, 1980
- Histochemical studies on the effect of (+)-amphetamine, drugs of the imipramine group and tryptamine on central catecholamine and 5-hydroxytryptamine neurons after intraventricular injection of catecholamines and 5-hydroxytryptamineEuropean Journal of Pharmacology, 1968
- Biochemical and Histochemical Studies on the Effects of Imipramine‐like Drugs and (+)‐Amphetamine on Central and Peripheral Catecholamine NeuronsActa Physiologica Scandinavica, 1966
- Anticonvulsant properties of some benzodiazepine derivativesNeurology, 1965