Oxidative stress, mitochondrial permeability transition and activation of caspases in calcium ionophore A23187-induced death of cultured striatal neurons
- 28 February 2000
- journal article
- Published by Elsevier in Brain Research
- Vol. 857 (1-2) , 20-29
- https://doi.org/10.1016/s0006-8993(99)02320-3
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Oxidative Stress in Huntington's DiseaseBrain Pathology, 1999
- Mitochondrial dysfunction in neurodegenerative diseasesBiochimica et Biophysica Acta (BBA) - Bioenergetics, 1998
- Glutamate Neurotoxicity in Rat Cerebellar Granule Cells: A Major Role for Xanthine Oxidase in Oxygen Radical FormationJournal of Neurochemistry, 1997
- Oxidative damage and metabolic dysfunction in Huntington's disease: Selective vulnerability of the basal gangliaAnnals of Neurology, 1997
- Chronic mitochondrial energy impairment produces selective striatal degeneration and abnormal choreiform movements in primates.Proceedings of the National Academy of Sciences, 1995
- Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acidJournal of Neuroscience, 1993
- Alternative excitotoxic hypothesesNeurology, 1992
- Immunohistochemical localization of DARPP-32 in striatal projection neurons and striatal interneurons: implications for the localization of D1-like dopamine receptors on different types of striatal neuronsBrain Research, 1991
- Replication of the neurochemical characteristics of Huntington's disease by quinolinic acidNature, 1986
- Growth of a rat neuroblastoma cell line in serum-free supplemented medium.Proceedings of the National Academy of Sciences, 1979