Alcohol causes both tolerance and sensitization of rat Kupffer cells via mechanisms dependent on endotoxin
- 1 August 1998
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 115 (2) , 443-451
- https://doi.org/10.1016/s0016-5085(98)70211-2
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Role of Calcium in Tumor Necrosis Factor- Production by Activated MacrophagesThe Journal of Biochemistry, 1996
- Cross‐Tolerance Between Acute Alcohol Intoxication and EndotoxemiaAlcohol, Clinical and Experimental Research, 1996
- Antibiotics prevent liver injury in rats following long-term exposure to ethanolGastroenterology, 1995
- Inactivation of Kupffer cells prevents early alcohol-induced liver injuryHepatology, 1994
- Alcohol Administration Attenuates LPS-Induced Expression of Inducible Nitric Oxide Synthase in Kupffer and Hepatic Endothelial CellsBiochemical and Biophysical Research Communications, 1993
- Effect of short-term ethanol treatment on voltage-dependent calcium channels in kupffer cellsHepatology, 1993
- Endotoxin hepatotoxicity augmented by ethanolExperimental and Molecular Pathology, 1991
- Biologically active products of stimulated liver macrophages (Kupffer cells)European Journal of Biochemistry, 1990
- Acute Alcohol Infusion Suppresses Endotoxin‐induced Serum Tumor Necrosis FactorAlcohol, Clinical and Experimental Research, 1989
- Biosynthesis of platelet-activating factor by cultured rat Kupffer cells stimulated with calcium ionophore A23187Biochemical Journal, 1989