Mitochondria Are Direct Targets of the Lipoxygenase Inhibitor MK886: A STRATEGY FOR CELL KILLING BY COMBINED TREATMENT WITH MK886 AND CYCLOOXYGENASE INHIBITORS

This publication has 47 references indexed in Scilit:

• Endonuclease G is an apoptotic DNase when released from mitochondria
  Nature, 2001
• Arachidonic Acid Causes Cell Death through the Mitochondrial Permeability Transition
  Journal of Biological Chemistry, 2001
• A mitochondrial perspective on cell death
  Trends in Biochemical Sciences, 2001
• Identification of DIABLO, a Mammalian Protein that Promotes Apoptosis by Binding to and Antagonizing IAP Proteins
  Cell, 2000
• Smac, a Mitochondrial Protein that Promotes Cytochrome c–Dependent Caspase Activation by Eliminating IAP Inhibition
  Cell, 2000
• Mitochondrial Transport of Cations: Channels, Exchangers, and Permeability Transition
  Physiological Reviews, 1999
• Bcl-2 inhibits the mitochondrial release of an apoptogenic protease.
  The Journal of Experimental Medicine, 1996
• Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome c
  Published by Elsevier ,1996
• Cyclooxygenase and lipoxygenase inhibitors in cancer therapy
  Prostaglandins, Leukotrienes & Essential Fatty Acids, 1996
• On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury
  Cardiovascular Research, 1993