A Single Transmembrane Site in the KCNE-encoded Proteins Controls the Specificity of KvLQT1 Channel Gating

This publication has 32 references indexed in Scilit:

• A common polymorphism associated with antibiotic-induced cardiac arrhythmia
  Proceedings of the National Academy of Sciences, 2000
• Maximal function of minimal K+ channel subunits
  Trends in Pharmacological Sciences, 2000
• Identification of Distinct Carboxyl-Terminal Domains Mediating Internalization and Down-Regulation of the Hamster α_1B- Adrenergic Receptor
  Molecular Pharmacology, 2000
• MiRP1 Forms IKr Potassium Channels with HERG and Is Associated with Cardiac Arrhythmia
  Published by Elsevier ,1999
• Single-Channel Characteristics of Wild-Type IKs Channels and Channels formed with Two MinK Mutants that Cause Long QT Syndrome
  The Journal of general physiology, 1998
• MinK-KvLQT1 Fusion Proteins, Evidence for Multiple Stoichiometries of the Assembled I Channel
  Journal of Biological Chemistry, 1998
• Voltage‐dependent inactivation of the human K⁺ channel KvLQT1 is eliminated by association with minimal K⁺ channel (minK) subunits
  The Journal of Physiology, 1998
• Mutations in the hminK gene cause long QT syndrome and suppress lKs function
  Nature Genetics, 1997
• The role of the I_sK protein in the specific pharmacological properties of the I_Ks channel complex
  British Journal of Pharmacology, 1997
• Alternative splicing of the human Shaker K⁺ channel β1 gene and functional expression of the β2 gene product
  FEBS Letters, 1995