Inhibition of Fas signaling prevents hepatic injury and improves organ blood flow during sepsis
- 1 August 2001
- Vol. 130 (2) , 339-345
- https://doi.org/10.1067/msy.2001.116540
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Differential Alterations in Cardiovascular Responses During the Progression of Polymicrobial Sepsis in the MouseShock, 2002
- Immune depression in polymicrobial sepsis: The role of necrotic (injured) tissue and endotoxinCritical Care Medicine, 2000
- ANTICYTOKINE THERAPIES FOR ACUTE INFLAMMATION AND THE SYSTEMIC INFLAMMATORY RESPONSE SYNDROME: IL-10 AND ISCHEMIA/REPERFUSION INJURY AS A NEW PARADIGMShock, 2000
- Apoptotic cell death in patients with sepsis, shock, and multiple organ dysfunctionCritical Care Medicine, 1999
- Is Fas Ligand or Endotoxin Responsible for Mucosal Lymphocyte Apoptosis in Sepsis?Archives of Surgery, 1998
- Heat Shock Proteins in the Regulation of the Apoptotic ResponseSepsis, 1998
- Sir Isaac Newton, sepsis, SIRS, and CARSCritical Care Medicine, 1996
- Regional blood flow and oxygen transport in septic shockCritical Care Medicine, 1993
- Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes.The Journal of Experimental Medicine, 1991
- Tumor necrosis factor and interleukin-1 serum levels during severe sepsis in humansCritical Care Medicine, 1989