Abnormal involuntary movements (AIMs) following pulsatile dopaminergic stimulation: Severe deterioration and morphological correlates following the loss of locus coeruleus neurons

This publication has 46 references indexed in Scilit:

• Synapse formation and plasticity: recent insights from the perspective of the ubiquitin proteasome system
  Current Opinion in Neurobiology, 2006
• Ubiquitin and Protein Turnover in Synapse Function
  Neuron, 2005
• Development of the dopaminergic neurons in the rodent brainstem
  Experimental Neurology, 2005
• Treatment with α2-adrenoceptor antagonist, 2-methoxy idazoxan, protects 6-hydroxydopamine-induced Parkinsonian symptoms in rats: neurochemical and behavioral evidence
  Behavioural Brain Research, 2004
• Behavioral and neurochemical effects of noradrenergic depletions with N-(2-chloroethyl)-N-ethyl-2-bromobenzylamine in 6-hydroxydopamine-induced rat model of Parkinson’s disease
  Behavioural Brain Research, 2004
• The ?2-adrenergic receptor antagonist idazoxan reduces dyskinesia and enhances anti-parkinsonian actions ofL-dopa in the MPTP-lesioned primate model of Parkinson's disease
  Movement Disorders, 1999
• Reduced MPTP neurotoxicity in striatum of the mutant mousetottering
  Synapse, 1998
• L‐DOPA‐induced dyskinesia in the rat is associated with striatal overexpression of prodynorphin‐ and glutamic acid decarboxylase mRNA
  European Journal of Neuroscience, 1998
• Clonidine Suppresses 1‐Methyl‐4‐Phenyl‐1,2,3,6‐Tetrahydropyridine‐Induced Reductions of Striatal Dopamine and Tyrosine Hydroxylase Activity in Mice
  Journal of Neurochemistry, 1995
• Locus coeruleus lesions potentiate neurotoxic effects of MPTP in dopaminergic neurons of the substantia nigra
  Brain Research, 1994