Time Course of the Development of Alzheimer-like Pathology in the Doubly Transgenic PS1+APP Mouse
- 1 February 2002
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 173 (2) , 183-195
- https://doi.org/10.1006/exnr.2001.7754
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Functional Phenotype in Transgenic Mice Expressing Mutant Human Presenilin-1Neurobiology of Disease, 2000
- Evidence for glial-mediated inflammation in aged APPSW transgenic miceNeurobiology of Aging, 1999
- Amyloid Phenotype Characterization of Transgenic Mice Overexpressing both Mutant Amyloid Precursor Protein and Mutant Presenilin 1 TransgenesNeurobiology of Disease, 1999
- Association of Microglia with Amyloid Plaques in Brains of APP23 Transgenic MiceThe American Journal of Pathology, 1999
- Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In VivoNeuron, 1996
- Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic MiceScience, 1996
- Increased amyloid-β42(43) in brains of mice expressing mutant presenilin 1Nature, 1996
- Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor proteinNature, 1995
- Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)Neuron, 1994
- The Pathogenesis and Progression of the Pathological Changes of Alzheimer's DiseaseAnnals of Medicine, 1989