Glucocorticoids Inhibit Apoptosis during Fibrosarcoma Development by Transcriptionally Activating Bcl-xL
Open Access
- 1 May 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (20) , 18022-18029
- https://doi.org/10.1074/jbc.m301812200
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and BakGenes & Development, 2001
- Glucocorticoid-Induced Apoptosis in LymphocytesBiochemical and Biophysical Research Communications, 2000
- The Hallmarks of CancerCell, 2000
- Both FGF1 and Bcl-x synthesis are necessary for the reduction of apoptosis in retinal pigmented epithelial cells by FGF2: role of the extracellular signal-regulated kinase 2Oncogene, 1999
- AIB1, a Steroid Receptor Coactivator Amplified in Breast and Ovarian CancerScience, 1997
- Glucocorticoid Repression of the Mouse Gonadotropin-releasing Hormone Gene Is Mediated by Promoter Elements That Are Recognized by Heteromeric Complexes Containing Glucocorticoid ReceptorPublished by Elsevier ,1996
- Transcription factors junB and c-jun are selectively up-regulated and functionally implicated in fibrosarcoma development.Genes & Development, 1992
- Social controls on cell survival and cell deathNature, 1992
- Dissecting Multistep Tumorigenesis in Transgenic MiceAnnual Review of Genetics, 1988
- Bovine papillomavirus genome elicits skin tumours in transgenic miceNature, 1986