Thymocyte depletion during acuteTrypanosoma cruziinfection in C57BL/6 mice is partly reverted by lipopolysaccharide pretreatment
Open Access
- 1 June 2004
- journal article
- Published by Oxford University Press (OUP) in FEMS Immunology & Medical Microbiology
- Vol. 41 (2) , 123-131
- https://doi.org/10.1016/j.femsim.2004.02.003
Abstract
Infection with Trypanosoma cruzi in C57BL/6 mice leads to a progressive fatal disease accompanied by thymocyte depletion, which is not related with aKeywords
This publication has 37 references indexed in Scilit:
- Interleukin-10 and the Interleukin-10 ReceptorAnnual Review of Immunology, 2001
- Administration of Endotoxin Associated with Lipopolysaccharide Tolerance Protects Mice against Fungal InfectionInfection and Immunity, 2000
- Regulatory T CellsCell, 2000
- Dissociation of IFN-γ from IL-12 and IL-18 production during endotoxin toleranceInnate Immunity, 1999
- Immunopathology of cardiomyopathy in the experimental Chagas diseaseMemórias do Instituto Oswaldo Cruz, 1999
- The cachexia associated with Trypanosoma cruzi acute infection in mice is attenuated by anti‐TNF‐a, but not by anti‐IL‐6 or anti‐IFN‐7 antibodiesParasite Immunology, 1995
- Lipopolysaccharide-induced selective priming effects on tumor necrosis factor alpha and nitric oxide production in mouse peritoneal macrophages.The Journal of Experimental Medicine, 1993
- The Pathophysiology of Tumor Necrosis FactorsAnnual Review of Immunology, 1992
- Adaptation to bacterial lipopolysaccharide controls lipopolysaccharide-induced tumor necrosis factor production in rabbit macrophages.Journal of Clinical Investigation, 1990
- Susceptible mice present higher macrophage activation than resistant mice during infections with myotropic strains of Trypanosoma cruziParasite Immunology, 1989