Overexpression of the Transcription Factor GATA-3 Enhances the Development of Pulmonary Fibrosis
- 1 July 2006
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 169 (1) , 96-104
- https://doi.org/10.2353/ajpath.2006.051160
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 53 references indexed in Scilit:
- Asthmatic changes in mice lacking T-bet are mediated by IL-13International Immunology, 2005
- GATA‐3 suppresses IFN‐γ promoter activity independently of binding to cis‐regulatory elementsFEBS Letters, 2004
- Transgenic Overexpression of GATA-3 in T Lymphocytes Improves Autoimmune Glomerulonephritis in Mice with a BXSB/MpJ-Yaa Genetic BackgroundJournal of the American Society of Nephrology, 2003
- Control of Regulatory T Cell Development by the Transcription Factor Foxp3Science, 2003
- LYMPHOKINES IN BLEOMYCIN-INDUCED LUNG INJURY IN BLEOMYCIN-SENSITIVE C57BL/6 AND-RESISTANT BALB/c MICEExperimental Lung Research, 2000
- Functional diversity of helper T lymphocytesNature, 1996
- Interleukin-4 and interferon-γ discordantly regulate collagen biosynthesis by functionally distinct lung fibroblast subsetsJournal of Cellular Physiology, 1996
- Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseasesImmunology Today, 1995
- Molecular Mechanisms of Antifibrotic Effect of Interferon Gamma in Bleomycin-Mouse Model of Lung Fibrosis: Downregulation of TGF-β and Procollagen I and III Gene ExpressionExperimental Lung Research, 1995
- Effect of Murine Gamma Interferon on the Cellular Responses to Bleomycin in MiceExperimental Lung Research, 1988