Engagement of CD44 by hyaluronan suppresses TLR4 signaling and the septic response to LPS
- 1 December 2009
- journal article
- Published by Elsevier in Molecular Immunology
- Vol. 47 (2-3) , 449-456
- https://doi.org/10.1016/j.molimm.2009.08.026
Abstract
No abstract availableKeywords
Funding Information
- NIH (P01HL057345, R01AR45676)
This publication has 52 references indexed in Scilit:
- NLRP3/Cryopyrin Is Necessary for Interleukin-1β (IL-1β) Release in Response to Hyaluronan, an Endogenous Trigger of Inflammation in Response to InjuryJournal of Biological Chemistry, 2009
- Electrophoresis studies on the contaminating glycosaminoglycans in commercially available hyaluronic acid productsElectrophoresis, 2008
- Hyaluronic acid: a natural biopolymer with a broad range of biomedical and industrial applicationsBiotechnology Letters, 2006
- Isolation, purification and characterization of hyaluronan from human umbilical cord residuesCarbohydrate Polymers, 2005
- Negative regulation of Toll-like receptor-mediated immune responsesNature Reviews Immunology, 2005
- Toll-like receptor signallingNature Reviews Immunology, 2004
- Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling PathwayScience, 2003
- Phylogenetic Perspectives in Innate ImmunityScience, 1999
- Molecular Basis of Endotoxin ToleranceaAnnals of the New York Academy of Sciences, 1998
- EFFECTS PRODUCED BY THE INTRAVENOUS INJECTION IN MAN OF A TOXIC ANTIGENIC MATERIAL DERIVED FROM EBERTHELLA TYPHOSA: CLINICAL, HEMATOLOGICAL, CHEMICAL AND SEROLOGICAL STUDIESJournal of Clinical Investigation, 1942