Long-term potentiation is associated with an increased activity of Ca2+/calmodulin-dependent protein kinase II.
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Open Access
- 4 January 2021
- journal article
- abstracts
- Published by Elsevier
- Vol. 268 (11) , 7863-7867
- https://doi.org/10.1016/s0021-9258(18)53037-4
Abstract
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This publication has 28 references indexed in Scilit:
- Deficient Hippocampal Long-Term Potentiation in α-Calcium-Calmodulin Kinase II Mutant MiceScience, 1992
- Activation of Ca2+/calmodulin-dependent protein kinase II in cerebellar granule cells by N-methyl-d-aspartate receptor activationMolecular and Cellular Neuroscience, 1990
- Inhibition of Postsynaptic PKC or CaMKII Blocks Induction But Not Expression of LTPScience, 1989
- An essential role for postsynaptic calmodulin and protein kinase activity in long-term potentiationNature, 1989
- Expression of a multifunctional Ca2+/calmodulin-dependent protein kinase and mutational analysis of its autoregulationPublished by Elsevier ,1989
- Multifunctional Ca2+/calmodulin-dependent protein kinase: domain structure and regulationTrends in Biochemical Sciences, 1989
- Immunohistochemical Localization of Ca2+/Calmodulin‐Dependent Protein Kinase II in Rat Brain and Various TissuesJournal of Neurochemistry, 1988
- Sequences of autophosphorylation sites in neuronal type II CaM kinase that control Ca2+-independent activityPublished by Elsevier ,1988
- Regulation of brain Type II Ca2+ calmodulin - dependent protein kinase by autophosphorylation: A Ca2+-triggered molecular switchPublished by Elsevier ,1986
- Translocation of Protein Kinase C Activity May Mediate Hippocampal Long-Term PotentiationScience, 1986