Stimulation of Primed Neutrophils by Soluble Immune Complexes: Priming Leads to Enhanced Intracellular Ca2+Elevations, Activation of Phospholipase D, and Activation of the NADPH Oxidase

This publication has 32 references indexed in Scilit:

• GTPγS-Stimulated Phospholipase D Activation in Human Neutrophils Occurs by Protein Kinase C-Dependent and -Independent Pathways but Not via Tyrosine Kinases
  Biochemical and Biophysical Research Communications, 1996
• ARF1‐regulated phospholipase D in human neutrophils is enhanced by PMA and MgATP
  FEBS Letters, 1994
• In Human Neutrophils the Binding to Immunocomplexes Induces the Tyrosine Phosphorylation of FcγRII But This Phosphorylation Is Not an Essential Signal for Fc-Mediated Phagocytosis
  Biochemical and Biophysical Research Communications, 1994
• Stimulation of reactive oxidant production in neutrophils by soluble and insoluble immune complexes occurs via different receptors/signal transduction systems
  FEMS Immunology & Medical Microbiology, 1994
• Receptor expression in synovial fluid neutrophils from patients with rheumatoid arthritis.
  Annals of the Rheumatic Diseases, 1993
• Granulocyte-macrophage colony-stimulating factor (GM-CSF) primes human neutrophils for enhanced phosphatidylcholine breakdown by phospholipase D
  Inflammation Research, 1991
• Neutrophils isolated from the synovial fluid of patients with rheumatoid arthritis: priming and activation in vivo.
  Annals of the Rheumatic Diseases, 1991
• Receptor expression and oxidase activity in human neutrophils: Regulation by granulocyte-macrophage colony-stimulating factor and dependence upon protein biosynthesis
  Bioscience Reports, 1990
• Phosphatidic acid and not diacylglycerol generated by phospholipase D is functionally linked to the activation of the NADPH oxidase by FMLP in human neutrophils
  Biochemical and Biophysical Research Communications, 1990
• Induction of neutrophil enzyme release by rheumatoid factors: Evidence for differences based on molecular characteristics
  Clinical Immunology and Immunopathology, 1986