Transient Diabetes Insipidus as a Complication of Haemophilus Meningitis

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Abstract
Diabetes insipidus has been described as a consequence of many acquired CNS lesions. It may develop following craniocerebral trauma,1 subarachnoid or intraventricular hemorrhage,2 hypothalamic lesions and pituitary tumors,3-5 leukemia,6 hypoxic encephalopathy,7 and neurohypophyseal inflammation.8 Although it is also reported as a late complication of tuberculous meningitis9-13 associated with suprasellar calcification, it most often occurs in this circumstance as part of the clinical picture of panhypopituitarism. The occurrence of diabetes insipidus, either temporary or permanent, as a complication of bacterial meningitis has rarely been reported. It is the purpose of this report to describe a case of transient vasopressin (Pitressin)-responsive diabetes insipidus complicating Haemophilus meningitis, to discuss the possible pathophysiology, and to outline the diagnosis and management.

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