Desensitization of the Permeability Transition Pore by Cyclosporin A Prevents Activation of the Mitochondrial Apoptotic Pathway and Liver Damage by Tumor Necrosis Factor-α

This publication has 63 references indexed in Scilit:

• Regulation of cell death: the calcium–apoptosis link
  Nature Reviews Molecular Cell Biology, 2003
• Apoptosis-based therapies
  Nature Reviews Drug Discovery, 2002
• TUMOR NECROSIS FACTOR RECEPTOR AND Fas SIGNALING MECHANISMS
  Annual Review of Immunology, 1999
• Caspase Cleaved BID Targets Mitochondria and Is Required for Cytochrome c Release, while BCL-XL Prevents This Release but Not Tumor Necrosis Factor-R1/Fas Death
  Journal of Biological Chemistry, 1999
• Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis
  Published by Elsevier ,1998
• Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors
  Cell, 1998
• Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria Blocked
  Science, 1997
• The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis
  Science, 1997
• Mice lacking the tumour necrosis factor receptor 1 are resistant to IMF-mediated toxicity but highly susceptible to infection by Listeria monocytogenes
  Nature, 1993
• Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection
  Cell, 1993