The TRAIL DISCussion: It is FADD and caspase-8!
- 30 August 2000
- journal article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 7 (9) , 759-760
- https://doi.org/10.1038/sj.cdd.4400735
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Apo2L/TRAIL-Dependent Recruitment of Endogenous FADD and Caspase-8 to Death Receptors 4 and 5Immunity, 2000
- Inherited Human Caspase 10 Mutations Underlie Defective Lymphocyte and Dendritic Cell Apoptosis in Autoimmune Lymphoproliferative Syndrome Type IICell, 1999
- The Death ReceptorsPublished by Springer Nature ,1999
- Targeted Disruption of the Mouse Caspase 8 Gene Ablates Cell Death Induction by the TNF Receptors, Fas/Apo1, and DR3 and Is Lethal PrenatallyImmunity, 1998
- FADD: Essential for Embryo Development and Signaling from Some, But Not All, Inducers of ApoptosisScience, 1998
- TRAIL Receptors 1 (DR4) and 2 (DR5) Signal FADD-Dependent Apoptosis and Activate NF-κBImmunity, 1997
- Death Receptor 5, a New Member of the TNFR Family, and DR4 Induce FADD-Dependent Apoptosis and Activate the NF-κB PathwayImmunity, 1997
- TRAIL-R2: a novel apoptosis-mediating receptor for TRAILThe EMBO Journal, 1997
- An Antagonist Decoy Receptor and a Death Domain-Containing Receptor for TRAILScience, 1997
- The Receptor for the Cytotoxic Ligand TRAILScience, 1997