Phenotypic variability within the JAK2 V617F-positive MPD: Roles of progenitor cell and neutrophil allele burdens
- 26 August 2008
- journal article
- research article
- Published by Elsevier in Experimental Hematology
- Vol. 36 (11) , 1480-1486.e2
- https://doi.org/10.1016/j.exphem.2008.05.006
Abstract
No abstract availableThis publication has 50 references indexed in Scilit:
- The JAK2 617V>F mutation triggers erythropoietin hypersensitivity and terminal erythroid amplification in primary cells from patients with polycythemia veraBlood, 2007
- Highly Sensitive Method for Genomewide Detection of Allelic Composition in Nonpaired, Primary Tumor Specimens by Use of Affymetrix Single-Nucleotide–Polymorphism Genotyping MicroarraysAmerican Journal of Human Genetics, 2007
- Molecular mimicry in the chronic myeloproliferative disorders: reciprocity between quantitative JAK2 V617F and Mpl expressionBlood, 2006
- Management of extreme thrombocytosis in otherwise low-risk essential thrombocythemia; does number matter?Blood, 2006
- Essential thrombocythemias without V617F JAK2 mutation are clonal hematopoietic stem cell disordersLeukemia, 2006
- A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia veraNature, 2005
- Heterogeneity of clonal development in chronic myeloproliferative disordersAmerican Journal of Hematology, 1999
- Clonal analysis of haemopoietic cells in essential thrombocythaemiaBritish Journal of Haematology, 1995
- Transient suppression of clonal hemopoiesis associated with pregnancy in a patient with a myeloproliferative disorder.Journal of Clinical Investigation, 1988
- Increased Incidence of Acute Leukemia in Polycythemia Vera Associated with Chlorambucil TherapyNew England Journal of Medicine, 1981